Macrophages just want to watch the whole body burn.
From what I’ve read, anything over about 101F is doing more harm than good.
That’s 38C for anyone out of the US
38C is a nice handful, not a temperature.
This does not appear to match the evidence anymore: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7717216/
The body is generally unable to raise the core temperature to the point where it can cause permanent damage, unless the ambient temperature is high. That is, unless there is a pre-existing heart or other vital organ condition.
In fact, fevers >=39C (102.2F) showed better outcomes in covid patients.
I thought danger was at 104F??
I only know ibuprofen as a pain killer, but good to know it can also reduce fever!
It was the key drug being deployed during the covid war. I survived tks to it. Saw god a few times.
Having a fever I can deal with, but I will immediately take some meds to not have a headache. Unfortunately, usually a fever comes with a headache.:(
To me Macrophages are the single most interesting creatures(symbiotes?) in the human body. I’ve read so much about them yet know so little.
Watch cells at work, you will thank me later
I’ve had this Excel workbook open for an hour now and my boss is asking why I’m just staring at it. But thank you?
It was an Excel joke.
you could say… it was a very good joke
You could say that he excelled at making jokes!
This sort of confusion just sheets me.
Ibuprofen does not reduce a fever, it reduces inflammation. Tylenol would have made this meme work
Edit: I was wrong
The first sentence on ibuprofens wikipedia page under “Medical uses”:
Ibuprofen is used primarily to treat fever
Anti-inflammatories reduce fevers, are there even anti-inflammatories that don’t have that effect to some extent?
So I was about to correct you that Tylenol is actually acetaminophen; turns out paracetamol is just another name for the same chemical. TIL
they both come from the chemical name - para-acetylamino-phenol (or, more proper, N-acetyl-para-aminophenol). random people chose different parts to shorten the name
Huh. I didn’t know that either.
I’ll be damned, I was almost certain it wasn’t an antipyretic, but it looks like I was wrong. Thanks for correcting me.
You didn’t need to use such condescending language though, there was no need to be rude.
Apologies. I do think making it sting a little when correcting someone on something they should have double checked, is warranted, if for no other reason than to make it clear to others reading which take to go home with.
But in response to your edit I’ve softened my correction as well.
Paracetamol is not anti-inflammatory in any serious context, which is to say taking paracetamol to reduce actual inflammation (think gout or rheumatoid arthritis) is more or less useless. From the wikipedia article on paracetamol:
Paracetamol inhibits prostaglandin synthesis by reducing the active form of COX-1 and COX-2 enzymes. This occurs only when the concentration of arachidonic acid and peroxides is low. Under these conditions, COX-2 is the predominant form of cyclooxygenase, which explains the apparent COX-2 selectivity of paracetamol. Under the conditions of inflammation, the concentration of peroxides is high, which counteracts the reducing effect of paracetamol. Accordingly, the anti-inflammatory action of paracetamol is slight.
It is, however, an analgesic.
Tylenol is arguably better but ibuprofen is also a fever reducer.
Nevermind that Tylenol’s effective ingredient is paracetamol, which is also “just” an anti-inflammatory.
Yeah I was just speaking about the fever reduction element but also true
Tylenol is a brand of acetaminophen, which is bad for your liver.
It’s metabolized by your liver, so is fat. It has a safe and therapeutic dosage range.
I am bad for my liver.
